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Jason Stoller, MD

Attending Neonatologist

Assistant Professor of Pediatrics, Perelman School of Medicine at the University of Pennsylvania Medical School

Original Papers

2013

Zhang T, Liu J, Zhang J, Thekkethottiyil EB, Macatee TL, Ismat FA, Wang F, Stoller JZ. Jun is required in Isl1-expressing progenitor cells for cardiovascular development. PLoS One. 2013;8(2):e57032. doi: 10.1371/journal.pone.0057032. Epub 2013 Feb 21. Read the abstract

2012 

Stoller JZ, Demauro SB, Dagle JM, Reese J. Current Perspectives on Pathobiology of the Ductus Arteriosus. J Clin Exp Cardiolog. 2012 Jun 15;8(1). pii: S8-001. Read the abstract

Posters and Presentations

2013

Pan H. Zhang T. De Mesmaeker J. Neve A, Benson MA, Latney BC, Werner P, Goldmuntz E, Bhattacharya S, Stoller, JZ. A Dominant Negative JUN mutant is associated with human congenital heart disease and alters a physicial and functional interaction with TBX1 (Platform Presentation). Weinsten Cardiovascular Development Conference. 2013 May 16-18, Tucson, AZ.

Pan H, Zhang T, Kraft CA, Subbaraj I, De Mesmaeker J, Latney BC, Goldmuntz E, Bhattacharya S, Stoller, JZ. TBX1 Interacts with JUN and a Dominant Negative JUN Missense Mutation Is Associated with Congenital Heart Disease (Platform Presentation). Eastern Society for Pediatric Research. 2013 Mar 22, Philadelphia, PA.

Pan H, Zhang T, Kraft CA, Subbaraj I, De Mesmaeker J, Latney BC, Goldmuntz E, Bhattacharya S, Stoller, JZ. TBX1 Interacts with JUN and a Dominant Negative JUN Missense Mutation Is Associated with Congenital Heart Disease (Platform Presentation). Pediatric Academic Societies (PAS) Annual Meeting. 2013 May 4-7, Washington DC.

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